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Your gut bacteria are your secret weight loss tool!! There has been a lot of hype surrounding the new GLP-1 agonist medications ,(Semaglutide, Wegovy, Ozempic, etc) due to their profound impact on weight loss. The reason that these drugs are so effective is due to their ability to stimulate cells within our gut to produce the hormone GLP-1, which is essentially a signal to your body that you are in a "fed state". This signal is detected by the pancreas, causing the release of insulin and the disposal of blood glucose (which is why the drug was initially meant to treat type 2 diabetes, due to the impact on blood glucose specifically). GLP-1 is also able to signal to the brain, which leads to a reduction in gastric emptying, and the slowing of digestion (which is one of the reasons people feel full so quickly on these medications)... The problem with these drugs, however, is the large proportion of people who experience significant side effects (mainly nausea and GI related distress, along with the potential to lose significant muscle mass). The side effect profiles are included in the clinical trial data (SURMOUNT, SURPASS, or SUSTAIN trials). What if I said you can have the same impact on GLP-1, but WITHOUT the drug and therefore without the side effects?!! Well guess what! YOU CAN!.. All you have to do is take care of your gut!! There are many strains and species of healthy bacteria lining our gut, and when we feed them dietary fiber (beans, vegetables, lentils, nuts, seeds, etc), they produce various short-chain fatty acids (butyrate, propionate, acetate). Those then go on to stimulate the gut to produce GLP-1!.. Which has the exact same effect that these pharmaceutical companies are going after!! Simply promoting a healthy gut microbiome with dietary fiber will result in an increase in GLP-1 secretion, and improved metabolic health. Here is a study exactly that (PMID: 29590046). Interested in learning more about the gut, and how it plays a role in our overall health? Tune in to the Kickback Science episode where we talk all about gut health: https://bit.ly/45jpJdf #gut #health #GLP1 #weightloss #metabolism #nutrition #fitness #science #research

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Amir Khan Fat Loss diet Aamir Khan, Aamir khan songs, Aamir khan Kapil Sharma show, Laal Singh Chaddha Aamir Khan Kapil Sharma's show, Aamir Khan Reveals His Secret Krushna Abhishek, Aamir khan, Aamir khan Kapil Sharma Lal Singh chadri Aamir khan Weight loss, Fitness Transformation, Bollywood, Health, Diet, Exercise, Workout, Inspiration, Motivation, Fat to fit, Celebrity, Lifestyle, Wellness, Nutrition and Diet Caloric Deficit: Consuming fewer calories than you burn. Macronutrients: Protein, carbohydrates, and fats—key diet components. High-Protein Diet: Eating more protein to preserve muscle mass during fat loss. Healthy Fats: Unsaturated fats from sources like avocados, nuts, and olive oil. Carbohydrate Cycling: Alternating high- and low-carb days. Intermittent Fasting: Eating within a specific time window (e.g., 16:8 fasting). Low-Calorie Foods: Foods with fewer calories per serving but high in volume (e.g., leafy greens). Metabolism: The rate at which your body burns calories for energy. Exercise and Physical Activity Strength Training: Building muscle to increase resting metabolic rate. Cardiovascular Exercise: Running, swimming, or cycling to burn calories. HIIT (High-Intensity Interval Training): Short, intense bursts of activity followed by rest periods. NEAT (Non-Exercise Activity Thermogenesis): Calories burned from daily activities (e.g., walking, fidgeting). Progressive Overload: Gradually increase the intensity of your workouts. Body Composition and Measurements Body Fat Percentage: The proportion of fat to total body weight. Lean Muscle Mass: Weight of muscles, bones, and organs excluding fat. Waist-to-Hip Ratio: A measure of fat distribution. BMI (Body Mass Index): A general measure of body weight with height. Lifestyle Factors Sleep Quality: Essential for recovery and hormonal balance. Stress Management: High stress can increase cortisol, leading to fat storage. Hydration: Drinking enough water for metabolism and fat oxidation. Consistency: Maintaining long-term adherence to a healthy lifestyle. Hormonal and Physiological Factors Insulin Sensitivity: Better glucose management for fat loss. Cortisol Levels: Stress hormone linked to abdominal fat. Leptin and Ghrelin: Hormones that regulate hunger and satiety. Thermogenesis: The production of heat in the body, which burns calories. Strategies and Tools Meal Prep: Preparing meals in advance to stay on track. Calorie Tracking: Using apps to monitor intake. Body Measurements: Regularly tracking progress (e.g., waist size). Supplements: Products like protein powders or caffeine (use with caution). Saket Gokhale, Cbum, Sam Sulek, Mr. Olympia, Amir Khan Fat Loss diet #healthtips #weightlosstips #Carb #tracking #caloricdeficit #weightloss

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New evidence brings more good news about the unanticipated effects of the GLP-1 receptor agonists. https://www.medscape.com/viewarticle/glp-1-drugs-might-cut-dementia-risk-half-2025a10008bs?src=soc_yt -- TRANSCRIPT -- Welcome to Impact Factor, your weekly dose of commentary on a new medical study. I’m Dr F. Perry Wilson from the Yale School of Medicine. It’s quite a thing that we live in an era with the biggest blockbuster weight-loss drugs in history, and I can look at you straight-faced and say, yeah, but honestly that’s not the most interesting thing about the GLP-1 receptor agonists such as Ozempic. Beyond their clear utility in weight loss, we have good data that demonstrate the drugs lead to improvements in rates of heart disease, liver disease, and even all-cause mortality, and intriguing reports of strange off-target effects: a reduction in gambling, drinking, drug use, smoking, and compulsive shopping. It’s honestly hard to find an organ system or self-destructive behavior that GLP-1 drugs don’t seem to improve somehow. It’s almost strange. It’s as if the statins came out and everyone was excited about the cholesterol going down, and then we discovered that they reversed hair loss or took three strokes off your golf game. How does one drug do so many things? I think the behavioral changes are the clue. The fact that people may drink or smoke less on these drugs should tell you that something is happening in their brains. And if these drugs are acting on the brain, you might well wonder if they can reduce the risk for brain disease — dementia. And, at least according to a new study, the answer appears to be yes. As the GLP-1 drugs started getting more and more popular, I decided to perform some due diligence and refresh what I had learned in med school about this thing called “glucagon-like peptide.” These drugs bind to the same receptor that GLP does, primarily to cause the pancreas to release more insulin when blood sugar is high. But we have plenty of drugs that stimulate insulin release. And they don’t seem to have all these other weird effects. So, what’s going on? When a drug acts on a specific receptor, it is worth seeing what cells in the body have that receptor, and this is a pretty interesting figure that shows that. You see high expression in the pancreas — no surprise. But next is expression in the heart muscle — interesting, considering these drugs have been shown to reduce the risk for heart disease. Then the salivary glands, and then… the cerebral cortex. The brain. And not just any part of the brain, the thinking part of the brain. A lot happens when these drugs bind to a receptor on a cell. But the brain is a mysterious and important organ. In contrast, the heart is relatively simple, and there are multiple randomized trials that show that GLP-1 drugs, as well as some other drugs for diabetes, such as the SGLT2 inhibitors, pioglitazone, and metformin, reduce the risk for cardiovascular disease in people with diabetes. So, it makes sense to ask whether these drugs would do the same for brain disease. Enter this study, "Cardioprotective Glucose-Lowering Agents and Dementia Risk," from Catriona Reddin and colleagues in JAMA Neurology. It’s an impressive bit of work combining data from 23 different randomized trials of these drugs including more than 150,000 individual people, to determine whether they reduce the risk for dementia. This is the best evidence we have to date for a neuroprotective effect of GLP-1 inhibitors. And this is the only type of study that could give us this evidence. The reason you need to combine so much data to detect a signal is that, to date, no randomized trials of GLP-1 drugs have been designed with dementia as a primary outcome. Transcript in its entirety can be found by clicking here: https://www.medscape.com/viewarticle/glp-1-drugs-might-cut-dementia-risk-half-2025a10008bs?src=soc_yt

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